P(doom) = 50%, 3 years to AGI.
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Currently upskilling for future work on technical AI alignment research. Want to do part time work translating AI developments in China for a Western audiance in the meantime. Would really appreciate funding on either.
Will analyze papers and do bioinformatics research for money. DM me if interested.
I can confirm that my PayPal has received the $500, although it'll be frozen for a while.
Thanks! I had a lot of fun doing the research for this and I'm working on an update that'll be out in a few days.
I think a lot of it comes down to training data context - " Leilan" is only present in certain videogame scrapes, " petertodd" is only found in Bitcoin spam, ect. So when you try to use it in a conversational context, the model starts spitting out weird stuff because it doesn't have enough information to understand what those tokens actually mean. I think GPT-2's guess for " petertodd" is something like "part of a name/email, if you see it, expect more mentions of Bitcoin". And not anything more, since that token doesn't occur much anywhere else. Thus, if you bring it up in a context where Bitcoin spam is very unlikely to occur, like a conversation with an AI assistant, it kinda just acts like a masked token, and you get the glitch token behavior.
I was thinking of areas along the gum-tooth interface having a local environment that normally promote tooth demineralization and cavities. After Lumina, that area could have high chronic acetaldehyde levels. In addition, the adaption of oral flora to the chronic presence of alcohol could increase first-pass metabolism, which increases acetaldehyde levels locally and globally during/after drinking.
I don't know how much Lumina changes the general oral environment, but I think you might be able to test this by seeing how much sugar you can put in your mouth before someone else can smell the fruity scent of acetaldehyde on your breath? I'm sure someone else can come up with a better experiment.
There are a lot of studies to regarding the assocation between ALDH2 deficiency and oral cancer risk. I think part of the issue is that
As always, biostatistics is hard! If X causes less drinking, drinking contributes to cancer, and X increases drinking's effects on cancer, X may have positive, neutral, or negative overall correlation with cancer. Most studies I've looked at had a pretty string correlation between ALDH2 deficiency and cancer though, especially after you control for alcohol consumption.
It also looks like most researchers in the field think the relationship is causal, with plausible mechanisms.
Will there be other people there? Looks like I'm the only one interested.
It might have conditioned your oral and gut flora to break down ethanol into acetaldehyde faster. I'll have a follow-up piece on hangovers and AFR coming soon, but the short of it is that certain antacids taken before drinking may help by decreasing ethanol first pass metabolism.
Good question - I don't think anyone knows.
Can you give examples of what you're looking for? Can I email you entries and expect a response?