We have no idea how to have a program detect AI-written text in a useful way.
This approach seems very doable:
I suppose you can call me lucky, but my wife and I had about two years of doing “quality of time spent” very well. And then we switched to building a family and that’s going well too. I guess you can have it all. 😊
Yup, I like it! Describes where I am pretty well.
I’ve been in the rationalist community since 2011. I too am focused on the “long path”. And I’d say my timelines are pretty short. But I have two young kids and I do not regret it. In fact we’ll probably have a third one.
Strongly agree with tangren
. Try to start interviewing and see if:
I will caution that right now is probably a particularly difficult time to find an engineering job. There were a lot of layoffs in big tech companies and a lot of them have a hiring freeze.
Excellent! Maybe there’s a way to pitch this for a Black Mirror episode.
I recently heard that it's possible that long-covid cough cause is actually neurological. Source: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8041436/ (Abstract below)
Has anyone heard about this? Anecdotally, the friend that told me about this took the required medicine and it cured his cough (which lasted 7 weeks up to that point) in half a day. This happened once he reached the right dose, which for him was 300 mg of gabapentin 2-3 times a day.
Quoting the abstract here:
Cough is one of the most common presenting symptoms of COVID-19, along with fever and loss of taste and smell. Cough can persist for weeks or months after SARS-CoV-2 infection, often accompanied by chronic fatigue, cognitive impairment, dyspnoea, or pain—a collection of long-term effects referred to as the post-COVID syndrome or long COVID. We hypothesise that the pathways of neurotropism, neuroinflammation, and neuroimmunomodulation through the vagal sensory nerves, which are implicated in SARS-CoV-2 infection, lead to a cough hypersensitivity state. The post-COVID syndrome might also result from neuroinflammatory events in the brain. We highlight gaps in understanding of the mechanisms of acute and chronic COVID-19-associated cough and post-COVID syndrome, consider potential ways to reduce the effect of COVID-19 by controlling cough, and suggest future directions for research and clinical practice. Although neuromodulators such as gabapentin or opioids might be considered for acute and chronic COVID-19 cough, we discuss the possible mechanisms of COVID-19-associated cough and the promise of new anti-inflammatories or neuromodulators that might successfully target both the cough of COVID-19 and the post-COVID syndrome.
This seems like a really good thing to do! I wish I could contribute more to the prize pool.
Wow, excellent advice all around. I’ve gone insane in exactly that way a few times, but later I learned that I have bipolar that gets triggered by stress and/or psychedelics. During the manic phase the mind runs away with whatever it’s thinking / obsessing about. Maybe that could potentially explain some of the other people too.
I don't think I define it rigorously. Maybe someone with deeper technical understanding of these models could.
But if I had to come up with a hack somehow, you could look at the distribution of probabilities for various words as ChatGPT is predicting the next token. Presumably you'll noticed a certain kind of probability distribution when it's in the "Luigi" mode and another when it's in "Waluigi" mode. Then prodding it in the right direction might be weighing more the tokens that are a lot more frequent in the Luigi mode than Waluigi.