Dear diary...
[this is an experiment in just posting little progress reports as a self-motivation tool.]
1. I have a growing suspicion that I was wrong to lump the amygdala in with the midbrain. It may be learning by the same reward signal as the neocortex. Or maybe not. It's confusing. Things I'm digesting: https://twitter.com/steve47285/status/1314553896057081857?s=19 (and references therein) and https://www.researchgate.net/publication/11523425_Parallels_between_cerebellum-_and_amygdala-dependent_conditioning
2. Speaking of mistakes, I'm also regretting some comments I made a while ago suggesting that the brain doesn't do backpropagation. Maybe that's true in a narrow sense, but Randall O'Reilly has convinced me that the brain definitely does error-driven learning sometimes (I already knew that), and moreover it may well be able to propagate errors through at least one or two layers of a hierarchy, with enough accuracy to converge. No that doesn't mean that the brain is exactly the same as a PyTorch / Tensorflow Default-Settings Deep Neural Net.
3. My long work-in-progress post on autism continues to be stuck on the fact that there seem to be two theories of social impairment that are each plausible and totally different. In one theory, social interactions are complex and hard to follow / model for cognitive / predictive-model-building reasons. The evidence I like for that is the role of the cerebellum, which sounds awfully causally implicated in autism. Like, absence of a cerebellum can cause autism, if I'm remembering right. In the other theory, modeling social interactions in the neurotypical way (via empathy) is aversive. The evidence I like for that is people with autism self-reporting that eye contact is aversive, among other things. (This is part of "intense world theory".) Of those two stories, I'm roughly 100% sold on the latter story is right. But the former story doesn't seem obviously wrong, and I don't like having two explanations for the same thing (although it's not impossible, autism involves different symptoms in different people, and they could co-occur for biological reasons rather than computational reasons). I'm hoping that the stories actually come together somehow, and I'm just confused about what the cerebellum and amygdala do. So I'm reading and thinking about that.
4. New theory I'm playing with: the neocortex outputs predictions directly, in addition to motor commands. E.g. "my arm is going to be touched". Then the midbrain knows not to flinch when someone touches the arm. That could explain why the visual cortex talks to the superior colliculus, which I always thought was weird. Jeff Hawkins says those connections are the neocortex sending out eye movement motor commands, but isn't that controlled by the frontal eye fields? Oh, then Randall O'Reilly had this mysterious throwaway comment in a lecture that the frontal eye fields seem to be at the bottom of the visual hierarchy if you look at the connections. (He had a reference, I should read it.) I don't know what the heck is going on.
Fair enough :-)